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Because a small change in the base current, IB, results in a large collector-current (IC) variation when the bias is just right, a transistor can operate as a current amplifier It might be more technically accurate to say that it is a current-fluctuation amplifier, because it s the magnification of current variations, not the absolute current, that s important

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We have elaborated the neuropsychiatric features of this disease, even though rare nowadays, because they are manifest almost uniquely as a chronic frontotemporoparietal meningoencephalitis and create a picture unlike that of most degenerative diseases discussed in Chap 39 with the possible exception of the newly popularized and imprecise category of frontotemporal dementia Also, it is well to remember that many of our ideas about the brain and the mind were shaped historically by this disease The blood serology is positive in nearly all cases The CSF is invariably abnormal, usually with 10 to 200 lymphocytes, plasma cells, and other mononuclear cells per cubic millimeter; a total protein of 40 to 200 mg/dL; an elevated gamma globulin; and strongly positive serologic tests The elevated gamma globulin in the CSF is produced intrathecally and has been shown to be adsorbed to T pallidum (Vartdal et al) Hence the gamma globulin (oligoclonal IgG) represents a speci c antibody response to this organism The pathologic changes consist of meningeal thickening, brain atrophy, ventricular enlargement, and granular ependymitis Microscopically, the perivascular spaces are lled with lymphocytes, plasma cells, and mononuclear cells; nerve cells have disappeared; there are numerous rod-shaped microgliacytes and plump astrocytes in parts of the cortex devastated by neuronal loss; iron is deposited in mononuclear cells; and, with special stains, spirochetes are visible in the cortex The changes are most pronounced in the frontal and temporal lobes The ependymal surfaces of the ventricles are studded with granular elevations protruding between ependymal cells (granular ependymitis) Meningeal brosis with obstructive hydrocephalus is present in many cases The prognosis in early treated cases has in the past been fairly good; 35 to 40 percent of patients made some occupational readjustment; in another 40 to 50 percent, the disease was arrested but left the patient dependent Without treatment there is progressive mental decline, and death occurs within 3 to 4 years Tabetic Neurosyphilis (Tabes Dorsalis) This type of neurosyphilis, described by Duchenne in his classic monograph L ataxie locomotrice progressive (1858), usually develops 15 to 20 years after the onset of the infection The major symptoms are lightning pains, ataxia, and urinary incontinence; the chief signs are absent tendon re exes at knee and ankle, impaired vibratory and position sense in feet and legs, and a Romberg sign The ataxia is due purely to the sensory defect Muscular power, by contrast, is fully retained in most cases The pupils are abnormal in over 90 percent of cases, usually Argyll-Robertson in type (see page 242), and the majority of patients show ptosis or some degree of ophthalmoplegia Optic atrophy is frequent The lancinating or lightning pains (present in over 90 percent of cases) are, as their name implies, sharp, stabbing, and brief, like a ash of lightning They are more frequent in the legs than elsewhere but roam over the body from face to feet, sometimes playing persistently on one spot like the repeated twanging of a ddle string, as Wilson remarked They may come in bouts lasting several hours or days Pins and needles feelings, coldness, numbness, tingling, and other paresthesias are also present and are associated invariably with impairment of tactile, pain, and thermal sensation The bladder is insensitive and hypotonic, resulting in unpredictable over ow incontinence Constipation and megacolon as well as impotence are other expressions of dysfunction of the sacral roots and ganglia In the established phase of the disease, now seldom seen, ataxia is the most prominent feature The patient totters and stag-.

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If you look at Fig 22-4 closely, you ll see that there are some bias values at which a transistor won t give current amplification If the E-B junction is not conducting, or if the transistor is in saturation, the curve is horizontal A small change (to the left and right) of the base voltage, EB, in these portions of the curve, will cause little or no up-and-down variation of IC But if the transistor is biased near the middle of the straight-line part of the curve in Fig 22-4, the transistor will work as a current amplifier

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